Bryan A. Sauls, MD, PhD

COPC Internal Medicine Group

Provider Overview

Accepting New Patients

Yes

Specialty

Internal Medicine

Languages Spoken

English

Practicing Since

2008

Provider Background

Areas of Interest

  • Preventative Care

  • Gastroenterology

  • Cardiology

Hospital Admitting Privileges

St. Ann's Hospital

Internships

The Ohio State University Medical Center

Residency

The Ohio State University Medical Center

Publications

  • Lenda, D., B. Sauls, and M. A. Boegehold. Local production of superoxide anion may contribute to reduced arteriolar nitric oxide in rats fed high salt. FASEB J. 13.4:A31, 1999; Sauls, B. A. and M. A. Boegehold.
  • A reduction in arteriolar wall PO2 may stimulates nitric oxide release during sympathetic vasoconstriction in the rat intestine. FASEB J. 13.4:A31, 1999; Sauls, B. A. and M. A. Boegehold.
  • Local adenosine production may stimulate arteriolar nitric oxide synthesis during sympathetic constriction in the rat intestine. FASEB J. 14.4:A3, 2000.; Sauls, B. A. and M. A. Boegehold.
  • Local PO2 reduction and adenosine release preserve arteriolar nitric oxide synthesis during sympathetic constriction in the rat intestine. J. Vasc. Res. 37(S1):32, 2000.; Sauls, B. A. and M. A. Boegehold.
  • Conversion to adenosine is not responsible for sustained endothelial nitric oxide synthesis during sympathetic vasoconstriction in the rat intestine. FASEB J. 15(4 pt 1):A103, 2001.; Lenda, D. M., B. A. Sauls, and M. A. Boegehold.
  • Reactive oxygen species may contribute to reduced endothelium-dependent dilation in rats fed high salt. Am. J. Physiol. Heart Circ. Physiol. 279:H7-H14, 2000.; Sauls, B. A. and M. A. Boegehold.
  • Arteriolar wall PO2 and nitric oxide release during sympathetic vasoconstriction in the rat intestine. Am. J. Physiol. Heart Circ. Physiol. 279:H484-H491, 2000.; Sauls, B. A. and M. A. Boegehold.
  • Reduced PO2 and adenosine formation preserve arteriolar nitric oxide synthesis during sympathetic constriction in the rat intestine. J. Vasc. Res. 38:104-112, 2001.; Sauls, B. A. and M. A. Boegehold.
  • Adenosine linking reduced O2 to arteriolar NO release in intestine is not formed from extracellular ATP. Am. J. Phsyiol. Heart Circ. Physiol. 281:H1193-H1200, 2001"

Additional Information

  • Certification in Basic Life Support and Advanced Cardiac Life Support

Medical School

Marshall University

Board Certification

American Board of Internal Medicine

Insurances Accepted

  • Aetna
  • Aetna Better Health aka Aetna MyCare
  • Aetna Medicare (COPC Senior Care Advantage Participant)
  • Anthem Blue Cross Blue Shield
  • Anthem Medicare (COPC Senior Care Advantage Participant)
  • BCMH
  • Buckeye Community Health Plan - Established Patients Only
  • Cigna
  • First Health
  • HealthSmart
  • Humana Commercial
  • Humana Medicare (COPC Senior Care Advantage Participant)
  • Medical Mutual of Ohio (MMO)
  • Medical Mutual OhioHealth HMO
  • Medicare
  • MediGold (COPC Senior Care Advantage Participant)
  • Meritain
  • Mt.Carmel Health Partners Trinity Health Plan
  • Multiplan
  • Ohio Health Choice (OHCP)
  • OhioHealthy
  • OSCAR Buckeye Health
  • OSU Health Plans
  • PHCS
  • PPO Connect
  • Railroad Medicare
  • Trinity Health Plan - Aetna Administered
  • UMWA
  • UnitedHealthcare

Patient resources

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Central Ohio Primary Care Logo

Bryan A. Sauls, MD, PhD

COPC Internal Medicine Group


Gender

Male

Specialty

Internal Medicine

Medical School

Marshall University

Board Certification

American Board of Internal Medicine

Practicing Since

2008

Languages Spoken

English

Hospital Admitting Privileges

St. Ann's Hospital

Accepting New Patients

Yes

Internships

The Ohio State University Medical Center

Residency

The Ohio State University Medical Center

Publications
  • Lenda, D., B. Sauls, and M. A. Boegehold. Local production of superoxide anion may contribute to reduced arteriolar nitric oxide in rats fed high salt. FASEB J. 13.4:A31, 1999; Sauls, B. A. and M. A. Boegehold.
  • A reduction in arteriolar wall PO2 may stimulates nitric oxide release during sympathetic vasoconstriction in the rat intestine. FASEB J. 13.4:A31, 1999; Sauls, B. A. and M. A. Boegehold.
  • Local adenosine production may stimulate arteriolar nitric oxide synthesis during sympathetic constriction in the rat intestine. FASEB J. 14.4:A3, 2000.; Sauls, B. A. and M. A. Boegehold.
  • Local PO2 reduction and adenosine release preserve arteriolar nitric oxide synthesis during sympathetic constriction in the rat intestine. J. Vasc. Res. 37(S1):32, 2000.; Sauls, B. A. and M. A. Boegehold.
  • Conversion to adenosine is not responsible for sustained endothelial nitric oxide synthesis during sympathetic vasoconstriction in the rat intestine. FASEB J. 15(4 pt 1):A103, 2001.; Lenda, D. M., B. A. Sauls, and M. A. Boegehold.
  • Reactive oxygen species may contribute to reduced endothelium-dependent dilation in rats fed high salt. Am. J. Physiol. Heart Circ. Physiol. 279:H7-H14, 2000.; Sauls, B. A. and M. A. Boegehold.
  • Arteriolar wall PO2 and nitric oxide release during sympathetic vasoconstriction in the rat intestine. Am. J. Physiol. Heart Circ. Physiol. 279:H484-H491, 2000.; Sauls, B. A. and M. A. Boegehold.
  • Reduced PO2 and adenosine formation preserve arteriolar nitric oxide synthesis during sympathetic constriction in the rat intestine. J. Vasc. Res. 38:104-112, 2001.; Sauls, B. A. and M. A. Boegehold.
  • Adenosine linking reduced O2 to arteriolar NO release in intestine is not formed from extracellular ATP. Am. J. Phsyiol. Heart Circ. Physiol. 281:H1193-H1200, 2001"


Additional Information
  • Certification in Basic Life Support and Advanced Cardiac Life Support