Provider Overview
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Practicing At
Accepting New Patients
No
Specialty
Internal Medicine
Languages Spoken
English
Practicing Since
2008
Provider Background
Areas of Interest
Preventive Medicine
Gastroenterology
Cardiology
Hospital Admitting Privileges
St. Ann's Hospital
Internships
The Ohio State University Medical Center
Residency
The Ohio State University Medical Center
Publications
- Lenda, D., B. Sauls, and M. A. Boegehold. Local production of superoxide anion may contribute to reduced arteriolar nitric oxide in rats fed high salt. FASEB J. 13.4:A31, 1999; Sauls, B. A. and M. A. Boegehold.
- A reduction in arteriolar wall PO2 may stimulates nitric oxide release during sympathetic vasoconstriction in the rat intestine. FASEB J. 13.4:A31, 1999; Sauls, B. A. and M. A. Boegehold.
- Local adenosine production may stimulate arteriolar nitric oxide synthesis during sympathetic constriction in the rat intestine. FASEB J. 14.4:A3, 2000.; Sauls, B. A. and M. A. Boegehold.
- Local PO2 reduction and adenosine release preserve arteriolar nitric oxide synthesis during sympathetic constriction in the rat intestine. J. Vasc. Res. 37(S1):32, 2000.; Sauls, B. A. and M. A. Boegehold.
- Conversion to adenosine is not responsible for sustained endothelial nitric oxide synthesis during sympathetic vasoconstriction in the rat intestine. FASEB J. 15(4 pt 1):A103, 2001.; Lenda, D. M., B. A. Sauls, and M. A. Boegehold.
- Reactive oxygen species may contribute to reduced endothelium-dependent dilation in rats fed high salt. Am. J. Physiol. Heart Circ. Physiol. 279:H7-H14, 2000.; Sauls, B. A. and M. A. Boegehold.
- Arteriolar wall PO2 and nitric oxide release during sympathetic vasoconstriction in the rat intestine. Am. J. Physiol. Heart Circ. Physiol. 279:H484-H491, 2000.; Sauls, B. A. and M. A. Boegehold.
- Reduced PO2 and adenosine formation preserve arteriolar nitric oxide synthesis during sympathetic constriction in the rat intestine. J. Vasc. Res. 38:104-112, 2001.; Sauls, B. A. and M. A. Boegehold.
- Adenosine linking reduced O2 to arteriolar NO release in intestine is not formed from extracellular ATP. Am. J. Phsyiol. Heart Circ. Physiol. 281:H1193-H1200, 2001"
Additional Information
- Certification in Basic Life Support and Advanced Cardiac Life Support
Medical School
Marshall University
Board Certification
American Board of Internal Medicine
Insurances Accepted
- Aetna
- Aetna Better Health aka Aetna MyCare
- Aetna Medicare (COPC Senior Health Connect Participant)
- Anthem Blue Cross Blue Shield
- Anthem Medicare (COPC Senior Health Connect Participant)
- BCMH
- Buckeye Community Health Plan - Established Patients Only
- Cigna
- First Health
- HealthSmart
- Humana Commercial
- Humana Medicare (COPC Senior Health Connect Participant)
- Medical Mutual of Ohio (MMO)
- Medical Mutual OhioHealth HMO
- Medicare
- MediGold (COPC Senior Health Connect Participant)
- Meritain
- Mt.Carmel Health Partners Trinity Health Plan
- Multiplan
- Ohio Health Choice (OHCP)
- OhioHealthy
- OSCAR Buckeye Health
- OSU Health Plans
- PHCS
- PPO Connect
- Railroad Medicare
- Trinity Health Plan - Aetna Administered
- UMWA
- UnitedHealthcare
Patient resources
Bryan A. Sauls, MD, PhD
COPC Internal Medicine Group
Gender
Male
Specialty
Internal Medicine
Medical School
Marshall University
Board Certification
American Board of Internal Medicine
Practicing Since
2008
Languages Spoken
English
Hospital Admitting Privileges
St. Ann's Hospital
Accepting New Patients
No
Internships
The Ohio State University Medical Center
Residency
The Ohio State University Medical Center
Publications
- Lenda, D., B. Sauls, and M. A. Boegehold. Local production of superoxide anion may contribute to reduced arteriolar nitric oxide in rats fed high salt. FASEB J. 13.4:A31, 1999; Sauls, B. A. and M. A. Boegehold.
- A reduction in arteriolar wall PO2 may stimulates nitric oxide release during sympathetic vasoconstriction in the rat intestine. FASEB J. 13.4:A31, 1999; Sauls, B. A. and M. A. Boegehold.
- Local adenosine production may stimulate arteriolar nitric oxide synthesis during sympathetic constriction in the rat intestine. FASEB J. 14.4:A3, 2000.; Sauls, B. A. and M. A. Boegehold.
- Local PO2 reduction and adenosine release preserve arteriolar nitric oxide synthesis during sympathetic constriction in the rat intestine. J. Vasc. Res. 37(S1):32, 2000.; Sauls, B. A. and M. A. Boegehold.
- Conversion to adenosine is not responsible for sustained endothelial nitric oxide synthesis during sympathetic vasoconstriction in the rat intestine. FASEB J. 15(4 pt 1):A103, 2001.; Lenda, D. M., B. A. Sauls, and M. A. Boegehold.
- Reactive oxygen species may contribute to reduced endothelium-dependent dilation in rats fed high salt. Am. J. Physiol. Heart Circ. Physiol. 279:H7-H14, 2000.; Sauls, B. A. and M. A. Boegehold.
- Arteriolar wall PO2 and nitric oxide release during sympathetic vasoconstriction in the rat intestine. Am. J. Physiol. Heart Circ. Physiol. 279:H484-H491, 2000.; Sauls, B. A. and M. A. Boegehold.
- Reduced PO2 and adenosine formation preserve arteriolar nitric oxide synthesis during sympathetic constriction in the rat intestine. J. Vasc. Res. 38:104-112, 2001.; Sauls, B. A. and M. A. Boegehold.
- Adenosine linking reduced O2 to arteriolar NO release in intestine is not formed from extracellular ATP. Am. J. Phsyiol. Heart Circ. Physiol. 281:H1193-H1200, 2001"
Additional Information
- Certification in Basic Life Support and Advanced Cardiac Life Support